AE
Abdullah Al Emran
Harvard Medical School, Centenary Institute
Research areas: Biochemistry, Genetics and Molecular Biology · Molecular Biology · Melanoma and MAPK Pathways · Epigenetics and DNA Methylation · Cancer Immunotherapy and Biomarkers
Employment
-
Harvard Medical School Research Fellow2021 - Present
-
Centenary Institute Postdoc2018 - Present
Education
Education history is unavailable.
Projects & Funding
Projects & funding information is unavailable.
Publications (32)
- BET inhibition sensitizes innate checkpoint inhibitor resistant melanoma to anti‐CTLA‐4 treatment Save
- Dual Targeting with EZH2 Inhibitor and STING Agonist to Treat Melanoma Save
- Nicotinamide Inhibits T Cell Exhaustion and Increases Differentiation of CD8 Effector T Cells Save
- Bioinformatics approaches to investigate the phytochemicals of Centella asiatica against the main protease of SARS-CoV-2 Save
- Repurposing Melanoma Chemotherapy to Activate Inflammasomes in the Treatment of BRAF/MAPK Inhibitor Resistant Melanoma. Save
- A Combination of Epigenetic BET and CDK9 Inhibitors for Treatment of Human Melanoma Save
- Lysine Demethylases: Promising Drug Targets in Melanoma and Other Cancers. Save
- DPP9: Comprehensive In Silico Analyses of Loss of Function Gene Variants and Associated Gene Expression Signatures in Human Hepatocellular Carcinoma Save
- Sex bias of females in survival from cancer and infections. Is X the answer? Save
- Do innate killing mechanisms activated by inflammasomes have a role in treating melanoma? Save
- Study of the Female Sex Survival Advantage in Melanoma—A Focus on X-Linked Epigenetic Regulators and Immune Responses in Two Cohorts Save
- EZH2 cooperates with DNA methylation to downregulate key tumour suppressors and interferon gene signatures in melanoma. Save
- Co-targeting bromodomain and extra-terminal proteins and MCL1 induces synergistic cell death in melanoma. Save
- Epigenetic inhibitors eliminate senescent melanoma BRAFV600E cells that survive long‑term BRAF inhibition. Save
- Pretreatment Innate Cell Populations and CD4 T Cells in Blood Are Associated With Response to Immune Checkpoint Blockade in Melanoma Patients. Save
- Commonly integrated epigenetic modifications of differentially expressed genes lead to adaptive resistance in cancer. Save
- Emerging roles of H3K9me3, SETDB1 and SETDB2 in therapy-induced cellular reprogramming. Save
- Targeting DNA Methylation and EZH2 Activity to Overcome Melanoma Resistance to Immunotherapy. Save
- Magnolol induces cell death through PI3K/Akt-mediated epigenetic modifications boosting treatment of BRAF- and NRAS-mutant melanoma. Save
- Distinct epigenetic modifications denote early drug resistance in cancer Save