Ravi Dhital
Nationwide Children's Hospital, Post Graduate Institute of Medical Education and Research
About
Ravi Dhital, PhD (Transplant Immunology), MSc (Human Immunology) is a research scientist in the Abigail Wexner Research institute at Nationwide Children’s Hospital (USA). His research focuses on study of Th1, Th17 and B- cells against viral infections in recipients with renal transplantation. He received his PhD from Post Graduate Institute of Medical Education and Research, India where he studied role of B- cells in allograft rejection and then moved to USA in 2017 for his post-doctoral research in the innate and adaptive immunity against Cytomegalovirus infection in renal transplant recipients. Of his more than 10 years of professional career, he had worked as a phlebotomist, histocompatibility technologist and a transplant immunologist. Currently, he is a member of the American Society of Transplantation, The Transplantation Society, American Society of histocompatibility and immunogenetics and American society of Nephrology. His linkedin profile is https://www.linkedin.com/in/ravi-dhital-a1670377/
Employment
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Nationwide Children's Hospital Research Scientist2022 - Present
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Nationwide Children's Hospital Postdoctoral Scientist2017 - 2021
Education
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Post Graduate Institute of Medical Education and Research M.S.2008 - 2010
Projects & Funding
Projects & funding information is unavailable.
Publications (7)
- Neopterin as a Biomarker of Cellular Immune Response in Renal Allograft Rejection Subtypes: Linking Cytokines and Immune Cells to Improve Diagnostic and Therapeutic Approaches Save
- Direct oHSV Infection Induces DC Maturation and a Tumor Therapeutic Response Save
- Oncolytic HSV-IL27 expression improves CD8 T cell function and therapeutic activity in syngeneic glioma models Save
- Virus-specific Th17 Cells Are Induced by Human Cytomegalovirus after Renal Transplantation Save
- T cell responses and clinical symptoms among infants with congenital cytomegalovirus infection Save
- Murine cytomegalovirus promotes renal allograft inflammation via Th1/17 cells and IL-17A Save
- Th1/17 Cells Infiltrate Murine Cytomegalovirus-Infected Renal Allografts via Virus-Induced CCL20 and Promote Th1 Cells through IL-17A Save