GR
Girish Radhakrishnan
National Institute of Animal Biotechnology, University of Wisconsin Madison, Madurai Kamaraj University
0000-0001-9088-0029
Employment
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National Institute of Animal Biotechnology Scientist2012 - Present
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University of Wisconsin Madison Research Faculty2011 - 2012
Education
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University of Wisconsin Madison Post-doctoral Research Associate2006 - 2010
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Madurai Kamaraj University PhD2000 - 2006
Projects & Funding
Projects & funding information is unavailable.
Publications (27)
- A Chimeric Peptide Derived from a Bacterial Effector Protein Attenuates TLR-2/4-Mediated Production of Pro-Inflammatory Cytokines and Enhances the Cellular Availability of Gentamicin Save
- The arginine/ornithine binding protein ArgT plays an essential role in Brucella neotomae / Brucella melitensis to prevent intracellular killing and contribute to chronic persistence in the host Save
- Brucella targets the host ubiquitin-specific protease, Usp8 , through the effector protein, TcpB, for facilitating infection of macrophages Save
- The arginine/ornithine binding protein ArgT plays an essential role inBrucellato prevent intracellular killing and contribute to chronic persistence in the host Save
- Brucellatargets host USP8 through the effector protein TcpB to facilitate infection of macrophages Save
- Host F-Box Protein 22 Enhances the Uptake of Brucella by Macrophages and Drives a Sustained Release of Proinflammatory Cytokines through Degradation of the Anti-Inflammatory Effector Proteins of Brucella Save
- Host F-box protein 22 enhances the uptake ofBrucellaby macrophages and drives a sustained release of pro-inflammatory cytokines through degradation of the anti-inflammatory effector proteins ofBrucella Save
- The neurosteroid pregnenolone promotes degradation of key proteins in the innate immune signaling to suppress inflammation Save
- Cytoplasmic Linker Protein CLIP170 Negatively Regulates TLR4 Signaling by Targeting the TLR Adaptor Protein TIRAP Save
- The Brucella effector protein TcpB induces degradation of inflammatory caspases and thereby subverts non-canonical inflammasome activation in macrophages Save