Tej Pratap Singh

Employment

• University of Pennsylvania
  2024 - Present
• GlaxoSmithKline (United States)
  2023 - 2024
• University of Pennsylvania
  2019 - 2023
• National Institute of Allergy and Infectious Diseases Research Fellow
  2016 - 2019
• National Institute of Allergy and Infectious Diseases Postdoctoral Fellow
  2011 - 2016
• Medical University of Graz Postdoctoral Fellow
  2010 - 2011
• Medical University of Graz Research Employee
  2007 - 2011
• National Institute of Malaria Research Senior Research Fellow
  2006 - 2007
• Institute of Genomics and Integrative Biology CSIR Project Research Fellow
  2004 - 2006

Education

• Medical University of Graz PhD
  2007 - 2010

Projects & Funding

Projects & funding information is unavailable.

Publications (36)

• Multiomic profiling of cutaneous leishmaniasis infections reveals microbiota-driven mechanisms underlying disease severity.
  Science translational medicine 2023 35 citations DOI: 10.1126/scitranslmed.adh1469
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• Regulatory T cells control Staphylococcus aureus and disease severity of cutaneous leishmaniasis
  Journal of Experimental Medicine 2023 26 citations DOI: 10.1084/jem.20230558
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• The skin microbiome enhances disease through IL-1b and delays healing in cutaneous leishmaniasis patients
  2023 4 citations DOI: 10.1101/2023.02.02.23285247
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• Human CCR6+ Th Cells Show Both an Extended Stable Gradient of Th17 Activity and Imprinted Plasticity.
  Journal of immunology (Baltimore, Md. : 1950) 2023 10 citations DOI: 10.4049/jimmunol.2200874
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• Editorial: Microbiota in skin inflammatory diseases.
  Frontiers in immunology 2023 4 citations DOI: 10.3389/fimmu.2023.1235314
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• Microbiota instruct IL-17A-producing innate lymphoid cells to promote skin inflammation in cutaneous leishmaniasis.
  PLoS pathogens 2021 28 citations DOI: 10.1371/journal.ppat.1009693
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• IL-23- and Imiquimod-Induced Models of Experimental Psoriasis in Mice
  Current protocols in immunology 2019 51 citations DOI: 10.1002/cpim.71
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• C/EBPδ drives interactions between human MAIT cells and endothelial cells that are important for extravasation
  eLife 2018 45 citations DOI: 10.7554/eLife.32532
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• Cbl-b deficiency provides protection against UVB-induced skin damage by modulating inflammatory gene signature
  Cell death & disease 2018 21 citations DOI: 10.1038/s41419-018-0858-5
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• Monocyte-derived inflammatory Langerhans cells and dermal dendritic cells mediate psoriasis-like inflammation
  Nature communications 2016 176 citations DOI: 10.1038/ncomms13581
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