Kameron B Rodrigues

Employment

• Stanford University Postdoctoral scholar
  2025 - Present
• Stanford University PhD Student
  2017 - 2024
• NIDDK/NIH Fellow
  2015 - 2017
• NCI/NIH Fellow
  2014 - 2015
• University of Arizona Student Researcher
  2013 - 2014

Education

• Stanford University PhD
  2017 - Present
• University of Arizona Bachelor of Science
  2010 - 2014

Projects & Funding

Projects & funding information is unavailable.

Publications (18)

• CD22-CAR T cell multiomic features linked to patient outcomes in CD19-CAR resistant large B cell lymphoma
  2026 DOI: 10.21203/rs.3.rs-7706791/v1
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• Exercise intensity and training alter the innate immune cell type and chromosomal origins of circulating cell-free DNA in humans
  Proceedings of the National Academy of Sciences 2025 DOI: 10.1073/pnas.2406954122
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• Deciphering the impact of genomic variation on function
  Writing group (ordered by contribution), J Engreitz, Heather A. Lawson +97 more
  Nature 2024 DOI: 10.1038/s41586-024-07510-0
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• Innate immune responses in the contexts of age-associated clonal hematopoiesis and regular exercise
  2024
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• Multi-Omic Profiling of Macrophages Lacking Tet2 or Dnmt3a Reveals Mechanisms of Hyper-Inflammation in Clonal Hematopoiesis
  Blood 2023 DOI: 10.1182/blood-2023-187890
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• Loss-of-function mutations in Dnmt3a and Tet2 lead to accelerated atherosclerosis and concordant macrophage phenotypes
  Nature Cardiovascular Research 2023 DOI: 10.1038/s44161-023-00326-7
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• The Impact of Genomic Variation on Function (IGVF) Consortium
  arXiv 2023
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• Transcriptomics and chromatin accessibility in multiple African population samples
  bioRxiv 2023 DOI: 10.1101/2023.11.04.564839
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• Powering Effective Climate Communication with a Climate Knowledge Base
  2021
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• Powering Effective Climate Communication with a Climate Knowledge Base
  arXiv: Computation and Language 2021
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• Innate immune stimulation of whole blood reveals IFN-1 hyper-responsiveness in type 1 diabetes
  Diabetologia 2020 DOI: 10.1007/s00125-020-05179-4
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• Restoration of the type I IFN-IL-1 balance through targeted blockade of PTGER4 inhibits autoimmunity in NOD mice
  JCI insight 2018 DOI: 10.1172/jci.insight.97843
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• PTGER4 inhibition in non-obese diabetic mice restores impaired IFNAR signaling, decreases chronic inflammatory signals and inhibits autoimmunity
  The Journal of Immunology 2017 DOI: 10.4049/jimmunol.198.supp.222.18
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• Belinostat and vincristine demonstrate mutually synergistic cytotoxicity associated with mitotic arrest and inhibition of polyploidy in a preclinical model of aggressive diffuse large B cell lymphoma.
  Cancer biology & therapy 2016 DOI: 10.1080/15384047.2016.1250046
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• Abstract 275: Combining low dose microtubule targeting agents with belinostat potentiates cytotoxic response in HDACi resistant diffuse large b-cell lymphoma
  Cancer Research 2016 DOI: 10.1158/1538-7445.am2016-275
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• Despite Increased Type 1 IFN, Autoimmune Nonobese Diabetic Mice Display Impaired Dendritic Cell Response to CpG and Decreased Nuclear Localization of IFN-Activated STAT1
  Journal of immunology (Baltimore, Md. : 1950) 2016 DOI: 10.4049/jimmunol.1501239
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• MET signaling in keratinocytes activates EGFR and initiates squamous carcinogenesis
  Science Signaling 2016 DOI: 10.1126/scisignal.aaf5106
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• Meta-analysis of crowdsourced data compendia suggests pan-disease transcriptional signatures of autoimmunity
  F1000Research 2016 DOI: 10.12688/f1000research.10465.1
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