AVIK DUTTA
National Institute of Allergy and Infectious Diseases, Children's National, SUNY Upstate Medical University, University of Calcutta Department of Biochemistry, University of Calcutta
About
I earned my B.S. and M.S. degrees in Biochemistry from the University of Calcutta, India, and received a Ph.D. from SUNY Upstate Medical University, USA. I have completed post-doctoral research at the University of Virginia, USA. Currently, I am a visiting postdoctoral Fellow at NICHD, NIH, USA. Skilled in Flow Cytometry, Confocal Imaging, Mouse Handling, Real-Time Polymerase Chain Reaction (qPCR), Microsoft Word, SDS-PAGE, Public Speaking, and Molecular Biology.
Employment
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National Institute of Allergy and Infectious Diseases Research Fellow2026 - Present
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Children's National Staff Scientist2025 - 2026
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National Institute of Child Health and Human Development Postdoctoral Research Fellow2020 - 2025
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University of Virginia Health System Postdoctoral Research Associate2019 - 2020
Education
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SUNY Upstate Medical University PhD2012 - 2018
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University of Calcutta Department of Biochemistry M.Sc2009 - 2011
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University of Calcutta B.Sc2006 - 2009
Projects & Funding
Projects & funding information is unavailable.
Publications (24)
- THEMIS: new insights into its evolution, structure, and function Save
- SRSF2 mutation reduces polycythemia and impairs hematopoietic progenitor functions in JAK2V617F-driven myeloproliferative neoplasm Save
- GRB2 promotes thymocyte positive selection by facilitating THEMIS-mediated inactivation of SHP1 Save
- THEMIS increases TCR signaling in CD4 + CD8 + thymocytes by inhibiting the activity of the tyrosine phosphatase SHP1 Save
- Genetic Deletion or Pharmacologic Inhibition of PTPN11 Impedes the Development and Progression of Myeloproliferative Neoplasms Induced By JAK2V617F and MPLW515L Mutants Save
- Interleukin-1 contributes to clonal expansion and progression of bone marrow fibrosis in JAK2V617F-induced myeloproliferative neoplasm Save
- Epigenetic Regulations of T Cell Differentiation Save
- Genetic ablation of Pim1 or pharmacologic inhibition with TP-3654 ameliorates myelofibrosis in murine models Save
- IL-1 Signaling Promotes Clonal Expansion and Progression of Bone Marrow Fibrosis in JAK2V617F-Induced Myeloproliferative Neoplasm Save
- U2af1 is required for survival and function of hematopoietic stem/progenitor cells Save