Harsh Goar
University of Texas Southwestern Medical Center, CSIR - Institute of Microbial Technology, Himachal Pradesh University
About
I am Harsh Goar, currently working as a Postdoctoral Fellow at University of Texas Southwestern Medical Center, Dallas, USA. I am trying to inhibit the histone synthesis as a cure for cancer.
I have recently finished my doctoral training. My dissertation entitled “Investigating into newer functions of virulence regulator PhoP: effect of PTMs on the functioning of the mycobacterial regulator”. My major training is on transcriptional regulation and integrated stress response of Mycobacterium tuberculosis which majorly involved molecular biology and microbiological techniques.
Employment
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University of Texas Southwestern Medical Center POSTDOC2022 - 2023
Education
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CSIR - Institute of Microbial Technology PhD2016 - 2022
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Himachal Pradesh University MSc
Projects & Funding
Projects & funding information is unavailable.
Publications (10)
- A Four Amino Acid Intracellular Motif of VISTA Blocks Growth Receptor Signaling in Cancer Cells to Induce Tumor Suppression Save
- Data from A Four Amino Acid Intracellular Motif of VISTA Blocks Growth Receptor Signaling in Cancer Cells to Induce Tumor Suppression Save
- Figure S1 from A Four Amino Acid Intracellular Motif of VISTA Blocks Growth Receptor Signaling in Cancer Cells to Induce Tumor Suppression Save
- Figure S2 from A Four Amino Acid Intracellular Motif of VISTA Blocks Growth Receptor Signaling in Cancer Cells to Induce Tumor Suppression Save
- Figure S3 from A Four Amino Acid Intracellular Motif of VISTA Blocks Growth Receptor Signaling in Cancer Cells to Induce Tumor Suppression Save
- Figure S4 from A Four Amino Acid Intracellular Motif of VISTA Blocks Growth Receptor Signaling in Cancer Cells to Induce Tumor Suppression Save
- Figure S5 from A Four Amino Acid Intracellular Motif of VISTA Blocks Growth Receptor Signaling in Cancer Cells to Induce Tumor Suppression Save
- Figure S6 from A Four Amino Acid Intracellular Motif of VISTA Blocks Growth Receptor Signaling in Cancer Cells to Induce Tumor Suppression Save
- Dual functioning by the PhoR sensor is a key determinant to Mycobacterium tuberculosis virulence Save
- Molecular Connectivity between Extracytoplasmic Sigma Factors and PhoP Accounts for Coupled Mycobacterial Stress Response Save