SANTANU DASGUPTA

About

I am a cancer researcher by choice, losing my beloved father due to cancer. Currently, I am a Tenured Associate Professor at the University of South Alabama and focused on translational cancer research aiming towards liquid biomarker development for early cancer detection, monitoring and guiding therapeutics. Earlier, I received training in mitochondrial genomics and biology in the laboratory of Dr. David Sidransky, Johns Hopkins School of Medicine.

Complete List of Published Work in My Bibliography (Total 88 publications):
https://www.ncbi.nlm.nih.gov/myncbi/1TcL2KNXilmki/bibliography/public/

Employment

• USA Mitchell Cancer Institute Associate Professor
  2025 - Present
• The University of Texas Health Science Center at Tyler Assistant Professor
  2013 - 2020

Education

• The PhD

Projects & Funding

Projects & funding information is unavailable.

Publications (38)

• Frequent Loss of CACNA1C Is Associated With Poor Prognosis in Non‐Small Cell Lung Cancer
  The FASEB Journal 2026 DOI: 10.1096/fj.202504868R
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• Genome-Wide DNA Methylation Profiling Reveals Ancestry-Associated Epigenetic Reprogramming in Cervical Intraepithelial Neoplasia
  International Journal of Molecular Sciences 2026 DOI: 10.3390/ijms27093986
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• The Current Roadmap of Lung Cancer Biology, Genomics and Racial Disparity
  International Journal of Molecular Sciences 2025 DOI: 10.3390/ijms26083818
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• Supplementary figure 7 from Mitochondrial Translocase TOMM22 Is Overexpressed in Pancreatic Cancer and Promotes Aggressive Growth by Modulating Mitochondrial Protein Import and Function
  2024 DOI: 10.1158/1541-7786.25125012.v1
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• Supplementary table 1 from Mitochondrial Translocase TOMM22 Is Overexpressed in Pancreatic Cancer and Promotes Aggressive Growth by Modulating Mitochondrial Protein Import and Function
  2024 DOI: 10.1158/1541-7786.25125009.v1
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• Supplementary table 1 from Mitochondrial Translocase TOMM22 Is Overexpressed in Pancreatic Cancer and Promotes Aggressive Growth by Modulating Mitochondrial Protein Import and Function
  2024 DOI: 10.1158/1541-7786.25125009
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• Supplementary figure 7 from Mitochondrial Translocase TOMM22 Is Overexpressed in Pancreatic Cancer and Promotes Aggressive Growth by Modulating Mitochondrial Protein Import and Function
  2024 DOI: 10.1158/1541-7786.25125012
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• Supplementary figure 6 from Mitochondrial Translocase TOMM22 Is Overexpressed in Pancreatic Cancer and Promotes Aggressive Growth by Modulating Mitochondrial Protein Import and Function
  2024 DOI: 10.1158/1541-7786.25125015
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• Supplementary figure 6 from Mitochondrial Translocase TOMM22 Is Overexpressed in Pancreatic Cancer and Promotes Aggressive Growth by Modulating Mitochondrial Protein Import and Function
  2024 DOI: 10.1158/1541-7786.25125015.v1
  +Save
• Supplementary figure 5 from Mitochondrial Translocase TOMM22 Is Overexpressed in Pancreatic Cancer and Promotes Aggressive Growth by Modulating Mitochondrial Protein Import and Function
  2024 DOI: 10.1158/1541-7786.25125018.v1
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