BB
Employment
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University of Pittsburgh Assistant Professor2020 - Present
Education
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University of Kansas Medical Center PhD2010 - 2016
Projects & Funding
Projects & funding information is unavailable.
Publications (67)
- Hepatocyte-Specific MET Deletion Exacerbates Acetaminophen-Induced Hepatotoxicity in Mice Save
- Hepatocyte-specific epidermal growth factor receptor (EGFR) deletion attenuates acetaminophen-induced liver injury in mice Save
- Diploid hepatocytes resist acetaminophen-induced liver injury through suppressed JNK signaling Save
- A Novel Role for the Phosphatidylinositol-4,5-Bisphosphate 3-Kinase Delta Isoform in Hepatocellular Proliferation Save
- Hepatocyte-Specific Deletion of EGFR Does Not Affect Steatosis But Enhances Fibrosis in a Fast-food Diet Model of MASLD Save
- Hepatocyte-specific fi c Epidermal Growth Factor Receptor Deletion Promotes Fibrosis but has no Effect on Steatosis in Fast-food Diet Model of Metabolic Dysfunction-associated Steatotic Liver Disease Save
- TARGETED DELETION OF EPIDERMAL GROWTH FACTOR RECEPTOR IN HEPATOCYTES MITIGATES ACETAMINOPHEN-INDUCED LIVER DAMAGE AND ENHANCES COMPENSATORY REGENERATION IN MICE Save
- Inhibiting Wnt Signaling Reduces Cholestatic Injury by Disrupting the Inflammatory Axis Save
- Regeneration and Recovery after Acetaminophen Hepatotoxicity Save
- HEPATOCYTE GROWTH FACTOR RECEPTOR (C-MET) IS REQUIRED FOR LIMITING HEPATOTOXICITY, PROMOTING LIVER REGENERATION AND RECOVERY FOLLOWING ACETAMINOPHEN-INDUCED ACUTE LIVER FAILURE IN MICE Save