DG
Domokos Gero
University of Exeter Medical School, University of Texas Medical Branch at Galveston, Semmelweis University, Medical School
Employment
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University of Exeter Medical School Marie Curie Research Fellow2015 - 2017
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University of Texas Medical Branch at Galveston Instructor (faculty)2010 - 2015
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University of Texas Medical Branch at Galveston Short term Scholar, Instructor2009 - 2009
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Semmelweis University, Medical School Associate Research Fellow2006 - 2010
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Semmelweis University, Medical School Assistant Research Fellow2004 - 2006
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Semmelweis University, Medical School PhD Trainee2000 - 2004
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Alexion Pharmaceuticals, Inc Intern1999 - 1999
Education
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Semmelweis University, Medical School Medical Doctor1994 - 2000
Projects & Funding
Projects & funding information is unavailable.
Publications (99)
- Individuality and information content of infrared molecular profiles: insights from a large longitudinal health-profiling study Save
- The novel mitochondria-targeted hydrogen sulfide (H 2 S) donors AP123 and AP39 protect against hyperglycemic injury in microvascular endothelial cells in vitro Save
- Development of a stretch-induced neurotrauma model for medium-throughput screening in vitro: identification of rifampicin as a neuroprotectant. Save
- Effect of endotoxemia in mice genetically deficient in cystathionine-γ-lyase, cystathionine-β-synthase or 3-mercaptopyruvate sulfurtransferase. Save
- Screening of a composite library of clinically used drugs and well-characterized pharmacological compounds for cystathionine β-synthase inhibition identifies benserazide as a drug potentially suitable for repurposing for the experimental therapy of colon… Save
- Glucocorticoids Suppress Mitochondrial Oxidant Production via Upregulation of Uncoupling Protein 2 in Hyperglycemic Endothelial Cells Save
- DEVELOPMENT OF A STRETCH-INDUCED NEUROTRAUMA MODEL FOR MEDIUM-THROUGHPUT SCREENING IN VITRO: IDENTIFICATION OF RIFAMPICIN AS A NEUROPROTECTANT Save
- EFFECT OF LIPOPOLYSACCHARIDE (LPS) - INDUCED ENDOTOXEMIA IN MICE DEFICIENT IN MICE GENETICALLY DEFICIENT IN EITHER ONE OF THE THREE MAJOR HYDROGEN-SULFIDE PRODUCING Save
- Hydrogen sulfide modulates chromatin remodeling and inflammatory mediator production in response to endotoxin, but does not play a role in the development of endotoxin tolerance. Save
- Mitochondrial slow-release hydrogen sulfide donors inhibit the hyperglycaemia-induced mitochondrial dysfunction Save