SP
Stefania Petrini
Bambino Gesù Children's Hospital, Istituto Ortopedico Rizzoli, Università degli Studi di Roma Tor Vergata Facoltà di Medicina e Chirurgia, Università degli Studi di Bologna
Research areas: Immunology and Microbiology · Immunology · Immune Cell Function and Interaction · Muscle Physiology and Disorders · Immunotherapy and Immune Responses
Employment
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Bambino Gesù Children's Hospital Biologo Ricercatore e Responsabile strutturato della Facility di Microscopia2011 - Present
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Bambino Gesù Children's Hospital Biologo Ricercatore e Responsabile della core Facility di Microscopia2007 - 2011
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Bambino Gesù Children's Hospital Ricercatore a contratto2000 - 2007
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Istituto Ortopedico Rizzoli Ricercatore a contratto1996 - 1998
Education
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Università degli Studi di Roma Tor Vergata Facoltà di Medicina e Chirurgia Laurea di Specializzazione in Patologia Clinica2006 - 2010
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Istituto Ortopedico Rizzoli Post-doctoral training1996 - 1996
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Università degli Studi di Bologna Dottorato di Ricercain Biologia Animale1992 - 1995
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Università degli Studi di Bologna Laurea in Scienze Biologiche1983 - 1991
Projects & Funding
Projects & funding information is unavailable.
Publications (168)
- Newly identified ARF3 variants strengthen the causal link between Golgi fragmentation and brain malformations Save
- Correction: Sennato et al. Optimization of Lipoplexes Functionalized with a Sialic Acid Mimetic (F9-PEG) to Target the C1858T PTPN22 Variant for Preclinical Assessment of a Novel Immunotherapy in Endocrine Autoimmunity. Pharmaceutics 2025, 17, 710 Save
- Optimization of Lipoplexes Functionalized with a Sialic Acid Mimetic (F9-PEG) to Target the C1858T PTPN22 Variant for Preclinical Assessment of a Novel Immunotherapy in Endocrine Autoimmunity Save
- Optimization of Lipoplexes Functionalized with a Sialic Acid Mimetic (F9-PEG) to Target the C1858T PTPN22 Variant for Preclinical Assessment of a Novel Immunotherapy in Endocrine Autoimmunity Save
- Imaging-Based Molecular Interaction Between Src and Lamin A/C Mechanosensitive Proteins in the Nucleus of Laminopathic Cells Save
- The Putative Role of TIM-3 Variants in Polyendocrine Autoimmunity: Insights from a WES Investigation Save
- The Putative Role Of TIM-3 Variants In Polyendocrine Autoimmunity: Insights From a WES Investigation Save
- Caspase-dependent apoptosis in Riboflavin Transporter Deficiency iPSCs and derived motor neurons Save
- Caspase-dependent apoptosis in Riboflavin Transporter Deficiency iPSCs and derived motor neurons Save
- Data from Nutlin-3a Enhances Natural Killer Cell–Mediated Killing of Neuroblastoma by Restoring p53-Dependent Expression of Ligands for NKG2D and DNAM-1 Receptors Save
- Data from Nutlin-3a Enhances Natural Killer Cell–Mediated Killing of Neuroblastoma by Restoring p53-Dependent Expression of Ligands for NKG2D and DNAM-1 Receptors Save
- Supplementary Figure legend from Nutlin-3a Enhances Natural Killer Cell–Mediated Killing of Neuroblastoma by Restoring p53-Dependent Expression of Ligands for NKG2D and DNAM-1 Receptors Save
- Supplementary Figure legend from Nutlin-3a Enhances Natural Killer Cell–Mediated Killing of Neuroblastoma by Restoring p53-Dependent Expression of Ligands for NKG2D and DNAM-1 Receptors Save
- Supplementary Figures from Nutlin-3a Enhances Natural Killer Cell–Mediated Killing of Neuroblastoma by Restoring p53-Dependent Expression of Ligands for NKG2D and DNAM-1 Receptors Save
- Supplementary Figures from Nutlin-3a Enhances Natural Killer Cell–Mediated Killing of Neuroblastoma by Restoring p53-Dependent Expression of Ligands for NKG2D and DNAM-1 Receptors Save
- Supplementary Table 1 from Nutlin-3a Enhances Natural Killer Cell–Mediated Killing of Neuroblastoma by Restoring p53-Dependent Expression of Ligands for NKG2D and DNAM-1 Receptors Save
- Supplementary Table 1 from Nutlin-3a Enhances Natural Killer Cell–Mediated Killing of Neuroblastoma by Restoring p53-Dependent Expression of Ligands for NKG2D and DNAM-1 Receptors Save
- Supplementary Video 1 from Nutlin-3a Enhances Natural Killer Cell–Mediated Killing of Neuroblastoma by Restoring p53-Dependent Expression of Ligands for NKG2D and DNAM-1 Receptors Save
- Supplementary Video 1 from Nutlin-3a Enhances Natural Killer Cell–Mediated Killing of Neuroblastoma by Restoring p53-Dependent Expression of Ligands for NKG2D and DNAM-1 Receptors Save
- SARS-CoV-2 infection of thymus induces loss of function that correlates with disease severity Save