Pramod Gupta

Employment

Employment history is unavailable.

Education

• Homi Bhabha National Institute Ph.D

Projects & Funding

Projects & funding information is unavailable.

Publications (14)

• Macrophage targeted polymeric curcumin nanoparticles limit intracellular survival of Mycobacterium tuberculosis through induction of autophagy and augment anti-TB activity of isoniazid in RAW 264.7 macrophages
  Frontiers in Immunology 2023 DOI: 10.3389/fimmu.2023.1233630
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• Biomarkers of Oxidative Stress and Its Dynamics in Cancer
  Handbook of Oxidative Stress in Cancer: Mechanistic Aspects 2022 DOI: 10.1007/978-981-15-9411-3_28
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• Quercetin induces proteolysis of mesenchymal marker vimentin through activation of caspase-3, and decreases cancer stem cell population in human papillary thyroid cancer cell line.
  Phytomedicine Plus 2021 DOI: 10.1016/j.phyplu.2021.100108
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• Host directed therapies: COVID-19 and beyond
  Current Research in Pharmacology and Drug Discovery 2021 DOI: 10.1016/j.crphar.2021.100058
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• Detection of Beijing strains of MDR M. tuberculosis and their association with drug resistance mutations in katG, rpoB, and embB genes
  BMC Infectious Diseases 2020 DOI: 10.1186/s12879-020-05479-5
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• New disease old vaccine: Is recombinant BCG vaccine an answer for COVID-19?
  Cellular Immunology 2020 DOI: 10.1016/j.cellimm.2020.104187
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• Intramacrophage Delivery of Dual Drug Loaded Nanoparticles for Effective Clearance of Mycobacterium tuberculosis
  Journal of Pharmaceutical Sciences 2020 DOI: 10.1016/j.xphs.2020.03.018
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• Polymeric curcumin nanoparticles by a facile in situ method for macrophage targeted delivery
  Bioengineering & Translational Medicine 2019 DOI: 10.1002/btm2.10112
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• Polysaccharide rich extract (PRE) from Tinospora cordifolia inhibits the intracellular survival of drug resistant strains of Mycobacterium tuberculosis in macrophages by nitric oxide induction
  Tuberculosis 2018 DOI: 10.1016/j.tube.2018.09.005
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• Activation of murine macrophages by G1-4A, a polysaccharide from Tinospora cordifolia, in TLR4/MyD88 dependent manner
  International Immunopharmacology 2017 DOI: 10.1016/j.intimp.2017.06.025
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