VC
Choudhary V
St. Jude Children's Research Hospital, Augusta University, Indian Veterinary Research Institute, Nagpur Veterinary College
Employment
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St. Jude Children's Research Hospital Manager- Lab Operations2024 - Present
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Augusta University Assistant Research Scientist2016 - 2024
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Augusta University Adjunct Instructor (Dermatoogy)2012 - 2016
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Augusta University Postdoctoral Fellow2010 - 2016
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Augusta University Postdoctoral Fellow2005 - 2010
Education
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Indian Veterinary Research Institute Ph.D.2000 - 2004
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Nagpur Veterinary College Masters in Veterinary Science (MVSc)1998 - 2000
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Nagpur Veterinary College Bachelor in Veterinary Science and Animal Husbandry (BVSc&AH), equivalent to DVM in USA1993 - 1998
Projects & Funding
Projects & funding information is unavailable.
Publications (40)
- The Effect of Glucocorticoid and Mineralocorticoid Receptor Antagonists in the Skin of Aged Female Mice Save
- Histone Deacetylase Inhibition Enhances AQP3 Levels in Human Corneal Epithelial Cells and Corneal Wound Healing in Normoglycemic and Diabetic Male Mice Save
- Exploring Skin Wound Healing Models and the Impact of Natural Lipids on the Healing Process Save
- Phosphatidylglycerol to Treat Chronic Skin Wounds in Diabetes Save
- Dioleoylphosphatidylglycerol Inhibits Heat Shock Protein B4 (HSPB4)-Induced Inflammatory Pathways In Vitro Save
- Advanced Glycation End Products and Activation of Toll-like Receptor-2 and -4 Induced Changes in Aquaporin-3 Expression in Mouse Keratinocytes Save
- Glycerol Improves Skin Lesion Development in the Imiquimod Mouse Model of Psoriasis: Experimental Confirmation of Anecdotal Reports from Patients with Psoriasis Save
- Pathogen-Associated Molecular Pattern-Induced TLR2 and TLR4 Activation Increases Keratinocyte Production of Inflammatory Mediators and is Inhibited by Phosphatidylglycerol. Save
- Dioleoylphosphatidylglycerol Accelerates Corneal Epithelial Wound Healing. Save
- Keratinocyte aquaporin-3 expression induced by histone deacetylase inhibitors is mediated in part by peroxisome proliferator-activated receptors (PPARs). Save