Mikhail Kolonin
University of Texas Medical School at Houston, University of Texas MD Anderson Cancer Center, Wayne State University
0000-0002-3743-7869
About
Mikhail Kolonin is a tenured Professor and the Harry E. Bovay, Jr. Distinguished University Chair in Metabolic Disease at the University of Houston Texas School of Medicine. He received his PhD in molecular medicine from Wayne State University where he pioneered the concept of expressing peptides designed to disrupt protein interactions in animals. As a post-doctoral fellow at M.D. Anderson Cancer Center, he has used screens of combinatorial libraries in mice and cancer patients to identify peptides targeting tissue-specific cell surface protein interactions. As a result, he has identified tissue-specific cell surface markers that have been pursued as drug targets. Dr. Kolonin has invented several experimental therapeutics targeting specific cell populations implicated in obesity and cancer. Current research of his laboratory converges on the multifaceted roles of progenitor cells from fat tissue in maintenance of healthy metabolism and in pathology. Currently, Dr. Kolonin is the Director of the Center for Metabolic and Degenerative Diseases at the Institute for Molecular Medicine.
Employment
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University of Texas Medical School at Houston Associate Professor2007 - Present
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University of Texas MD Anderson Cancer Center postdoctoral fellow / Instructor2000 - 2007
Education
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Wayne State University PhD1994 - 2000
Projects & Funding
Projects & funding information is unavailable.
Publications (89)
- Fibulin-4 expressed in metastatic breast cancer is a target of peptide-based imaging probes and experimental therapeutics Save
- GLP1 Mimetics and Stem Cells: An Intersection of Metabolic and Regenerative Medicine Save
- Adipose Stromal Cell-Derived Cancer-Associated Fibroblasts Promote Pancreatic Adenocarcinoma Progression Through SFRP4 Signaling Save
- Lipids grease the chain of cancer progression Save
- Adipocyte-specific deletion of sine oculis homeobox homolog 1 inhibits lipolysis and reduces skin fibrosis Save
- Telomerase Knockout in Myeloid Cells Predisposes Mice to Foam Cell Formation, Dyslipidemia, Lung Fibrosis, and Cardiac Dysfunction Save
- Figure 2 from Depletion of Adipose Stroma-Like Cancer-Associated Fibroblasts Potentiates Pancreatic Cancer Immunotherapy Save
- Depletion of Adipose Stroma-Like Cancer-Associated Fibroblasts Potentiates Pancreatic Cancer Immunotherapy Save
- Figure S3 from Depletion of Adipose Stroma-Like Cancer-Associated Fibroblasts Potentiates Pancreatic Cancer Immunotherapy Save
- Figure S2 from Depletion of Adipose Stroma-Like Cancer-Associated Fibroblasts Potentiates Pancreatic Cancer Immunotherapy Save