KZ
Kai Zhao
Central South University, Chinese Academy of Medical Sciences & Peking Union Medical College
Research areas: Immunology and Microbiology · Immunology · Immune Response and Inflammation · Inflammasome and immune disorders · Hematopoietic Stem Cell Transplantation
0000-0002-3889-2118
Employment
- 2017 - Present
Education
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Chinese Academy of Medical Sciences and Peking Union Medical College Doctor of Medicine2014 - 2017
Projects & Funding
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YAP调控NLRP3炎性小体活化参与脓毒症休克的机制研究 National Natural Science Foundation of China2019 - 2021
Publications (11)
- Author Correction: STAT3 promotes NLRP3 inflammasome activation by mediating NLRP3 mitochondrial translocation Save
- STAT3 promotes NLRP3 inflammasome activation by mediating NLRP3 mitochondrial translocation Save
- Acetylation is required for full activation of the NLRP3 inflammasome Save
- Leukadherin-1 inhibits NLRP3 inflammasome by blocking inflammasome assembly Save
- Z-DNA binding protein 1 promotes heatstroke-induced cell death Save
- β-catenin promotes NLRP3 inflammasome activation via increasing the association between NLRP3 and ASC Save
- Acetylation is required for NLRP3 self-aggregation and full activation of the inflammasome Save
- Caspse-11-GSDMD pathway is required for serum ferritin secretion in sepsis Save
- The complement receptor C5aR2 promotes protein kinase R expression and contributes to NLRP3 inflammasome activation and HMGB1 release from macrophages Save
- Cytoplasmic STAT4 Promotes Antiviral Type I IFN Production by Blocking CHIP-Mediated Degradation of RIG-I Save
- Tet2 is required to resolve inflammation by recruiting Hdac2 to specifically repress IL-6 Save