SR
Shayaq Ul Abeer Rasool
Also known as: Dr. Shayaq
Sher-i-Kashmir Institute of Medical Sciences, Sher-E-Kashmir University of Agricultural Sciences and Technology Kashmir, University of Kashmir
Employment
-
Sher-i-Kashmir Institute of Medical Sciences Scientist II2023 - Present
-
Government Degree College Lecturer2022 - 2022
-
Sher-E-Kashmir University of Agricultural Sciences and Technology Kashmir Research Fellow2020 - Present
-
University of Kashmir Research Scholar
Education
-
University of Kashmir M.Phil2012 - 2015
-
University of Kashmir MSC
Projects & Funding
Projects & funding information is unavailable.
Publications (23)
- Promoter hypermethylation drives a paradoxical up regulation of hTERT with prognostic implications in bladder cancer Save
- Significant Implications Of miR-126 Expression With Respect to Its Target Gene VEGFA in the Pathogenesis of Recurrent Pregnancy Losses (RPL)-a Study in High Incidence Zone Save
- IL-11–IL6ST–STAT3 signaling defines a convergent inflammatory axis in esophageal cancer subtypes Save
- HLA-G Polymorphic Variation (G*0104N exon 3) Confers Potential Risk for Recurrent Pregnancy Losses: A Study in a High Incidence Zone (Kashmir, North India) Save
- Implications of lipoprotein (a) [Lp(a)] gene polymorphic sequence variations and its protein expression in venous thromboembolism (VTE) Save
- Overexpression of androgen receptor signalling transactivator lncRNAs PRNCR1 and PCGEM1 are associated with the increased risk of PCOS Save
- Polymorphic Variants of FOXP3 Gene (rs 3761548) and (rs 3761549) are Significantly Associated with the Risk for Recurrent Pregnancy Losses. A Study in High Incidence Zone (Kashmir, North India) Save
- Inflammatory mediator profile of hospitalised patients with COVID‑19 during the second wave of the pandemic: A comparative study Save
- Significance and implications of FHIT gene expression and promoter hypermethylation in acute lymphoblastic leukemia (ALL) Save
- Androgen receptor signalling transactivator lncRNAs PRNCR1 and PCGEM contribute to PCOS pathogenesis Save