Esteban Chacón-Solano

Employment

• Carlos III University of Madrid Assistant Professor
  2023 - Present

Education

• Universidad Carlos III de Madrid PhD in Biomedical Science and Technology
  2016 - 2022
• Universidad Carlos III de Madrid MSc Biomedical Technologies Management and Development
  2015 - 2016
• Instituto Tecnológico de Costa Rica Biotechnology Engineering
  2010 - 2015

Projects & Funding

Projects & funding information is unavailable.

Publications (18)

• 368 Epithelial-mesenchymal crosstalk communication promotes fibroblast activation and fibrosis in RDEB
  Journal of Investigative Dermatology 2023 DOI: 10.1016/j.jid.2023.03.373
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• FPR2 DNA Aptamers for Targeted Therapy of Wound Repair
  Journal of Investigative Dermatology 2022 DOI: 10.1016/j.jid.2021.12.026
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• Mechanistic interrogation of mutation-independent disease modulators of RDEB identifies the small leucine-rich proteoglycan PRELP as a TGF-β antagonist and inhibitor of fibrosis
  Matrix Biology 2022 DOI: 10.1016/j.matbio.2022.06.007
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• Read-Through for Nonsense Mutations in Type XVII Collagen‒Deficient Junctional Epidermolysis Bullosa
  Journal of Investigative Dermatology 2022 DOI: 10.1016/j.jid.2021.09.018
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• The RareDis corpus: A corpus annotated with rare diseases, their signs and symptoms
  Journal of Biomedical Informatics 2022 DOI: 10.1016/j.jbi.2021.103961
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• 164 Molecular characterization of the response to haploidentical mesenchymal stem cells treatment in recessive dystrophic epidermolysis bullosa patients
  Journal of Investigative Dermatology 2021 DOI: 10.1016/j.jid.2021.08.168
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• 361 Identification of a new molecular modulator of severity in recessive dystrophic epidermolysis bullosa
  Journal of Investigative Dermatology 2021 DOI: 10.1016/j.jid.2021.08.370
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• Correction of recessive dystrophic epidermolysis bullosa by homology-directed repair-mediated genome editing
  Jose Bonafont, Ángeles Mencía, Esteban Chacón-Solano +11 more
  Molecular Therapy 2021 DOI: 10.1016/j.ymthe.2021.02.019
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• Efficient CRISPR-Cas9-Mediated Gene Ablation in Human Keratinocytes to Recapitulate Genodermatoses: Modeling of Netherton Syndrome
  Molecular Therapy Methods and Clinical Development 2020 DOI: 10.1016/j.omtm.2020.05.031
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• Humanization of tumor stroma by tissue engineering as a tool to improve squamous cell carcinoma xenograft
  International Journal of Molecular Sciences 2020 DOI: 10.3390/ijms21061951
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• 131 Phase I/II efficacy and safety study of mesenchymal stromal cells in recessive dystrophic epidermolysis bullosa
  Journal of Investigative Dermatology 2019 DOI: 10.1016/j.jid.2019.07.135
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• 309 Development of a Netherton Syndrome model based on targeted deletion of SPINK5 in human keratinocytes
  Journal of Investigative Dermatology 2019 DOI: 10.1016/j.jid.2019.07.310
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• 631 Oxidative stress imbalance as contributing factor in the establishment of fibrosis in recessive dystrophic epidermolysis bullosa
  Journal of Investigative Dermatology 2019 DOI: 10.1016/j.jid.2019.07.636
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• Clinically Relevant Correction of Recessive Dystrophic Epidermolysis Bullosa by Dual sgRNA CRISPR/Cas9-Mediated Gene Editing
  Molecular Therapy 2019 DOI: 10.1016/j.ymthe.2019.03.007
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• Fibroblast activation and abnormal extracellular matrix remodelling as common hallmarks in three cancer-prone genodermatoses
  British Journal of Dermatology 2019 DOI: 10.1111/bjd.17698
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• Organotypic and humanized animal models of genodermatoses
  Skin Tissue Models 2018 DOI: 10.1016/B978-0-12-810545-0.00004-8
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• 116 Comparative transcriptomic analysis of fibroblasts from two sisters with discordant severe generalized recessive dystrophic epidermolysis bullosa phenotype reveals new molecular markers associated with disease severity
  Journal of Investigative Dermatology 2017 DOI: 10.1016/j.jid.2017.07.426
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• 601 Common mechanisms and signaling pathways in fibroblasts of cancer-prone genodermatoses
  Journal of Investigative Dermatology 2017 DOI: 10.1016/j.jid.2017.07.798
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