MR
Mari Carmen Romero-Mulero
ETH Zurich, Max Planck Institute of Immunobiology and Epigenetics, Universitat Autònoma de Barcelona, Universidad de Sevilla
Research areas: Medicine · Hematology · Hematopoietic Stem Cell Transplantation · Mesenchymal stem cell research · Acute Myeloid Leukemia Research
Employment
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ETH Zürich Postdoctoral researcher2025 - Present
- 2020 - 2025
Education
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Universitat Autònoma de Barcelona Master Degree in Bioinformatics2019 - 2020
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Universidad de Sevilla Bachelor Degree in Biochemistry2015 - 2019
Projects & Funding
Projects & funding information is unavailable.
Publications (13)
- Multimodal omics identifies a lymphoid-biased multipotent progenitor population with limited age-associated remodeling Save
- Differentiation, ageing and leukaemia alter the metabolic profile of human bone marrow haematopoietic stem and progenitor cells Save
- Modulation of bone marrow haematopoietic stem cell activity as a therapeutic strategy after myocardial infarction: a preclinical study Save
- Chromatin landscape at cis-regulatory elements orchestrates cell fate decisions in early embryogenesis Save
- GPRC5C drives branched-chain amino acid metabolism in leukemogenesis Save
- A Journey Into the Unknown: PhD Students in a European Training Network on Age-related Changes in Hematopoiesis Conduct Their Project During a Global Pandemic Save
- Hyaluronic acid–GPRC5C signalling promotes dormancy in haematopoietic stem cells Save
- Multilayer omics analysis reveals a non-classical retinoic acid signaling axis that regulates hematopoietic stem cell identity Save
- Prenatal inflammation perturbs fetal hematopoietic development and causes persistent changes to postnatal immunity Save
- Prenatal inflammation perturbs murine fetal hematopoietic development and causes persistent changes to postnatal immunity Save
- Mouse multipotent progenitor 5 cells are located at the interphase between hematopoietic stem and progenitor cells Save
- ULK1 and ULK2 are less redundant than previously thought: computational analysis uncovers distinct regulation and functions of these autophagy induction proteins. Save
- ULK1 and ULK2 are less redundant than previously thought: Computational analysis uncovers distinct regulation and functions of these autophagy induction proteins Save