RG
Ruchi Gera
Karolinska Institutet, Karolinska University Hospital, Yale University
Research areas: Neuroscience · Cellular and Molecular Neuroscience · Neuroinflammation and Neurodegeneration Mechanisms · Alzheimer's disease research and treatments · Cholinesterase and Neurodegenerative Diseases
Employment
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Karolinska Institute Postdoctoral Researcher2020 - Present
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Yale University Postdoctoral Associate2019 - 2020
Education
Education history is unavailable.
Projects & Funding
Projects & funding information is unavailable.
Publications (14)
- CTLA-4 blockade shifts the B cell repertoire toward autoimmunity Save
- Positive and negative selection shape the human naive B cell repertoire Save
- Chronic exposure to Pb 2+ perturbs ChREBP transactivation and coerces hepatic dyslipidemia Save
- Sneaky Entry of IFNγ Through Arsenic-Induced Leaky Blood–Brain Barrier Reduces CD200 Expression by Microglial pro-Inflammatory Cytokine Save
- Exposure to arsenic promotes thymocyte commitment into CD4 lineage and induces immunosuppression by elevating regulatory T cells Save
- Zinc oxide nanoparticle induces microglial death by NADPH-oxidase-independent reactive oxygen species as well as energy depletion Save
- Arsenic exposure impels CD4 commitment in thymus and suppress T cell cytokine secretion by increasing regulatory T cells Save
- Fluorometric Estimation of Glutathione in Cultured Microglial Cell Lysate Save
- Arsenic exposure alters thymocyte lineage commitment and suppresses splenic T cell function by increasing regulatory T cells Save
- Hijacking microglial glutathione by inorganic arsenic impels bystander death of immature neurons through extracellular cystine/glutamate imbalance Save
- Immunomodulation by Environmental Chemicals: Insights into Mammalian Immune Responses to Arsenic, Cadmium, and Lead Save
- Isolation and characterization of microglia from adult mouse brain: selected applications for ex vivo evaluation of immunotoxicological alterations following in vivo xenobiotic exposure Save
- Comparative toxicity of low dose tributyltin chloride on serum, liver, lung and kidney following subchronic exposure Save
- Tributyltin induces oxidative damage, inflammation and apoptosis via disturbance in blood–brain barrier and metal homeostasis in cerebral cortex of rat brain: an in vivo and in vitro study Save