0000-0002-7367-5848 Employment
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Publications (16)
- Heavy metal toxicity as a driver of endoplasmic reticulum stress and dysfunction Save
- Disruption of Lysosomal Homeostasis Following Combined Exposure to Lead and Amyloid‐β Peptides (25–35) and (1–40) in SH‐SY5Y Cells Save
- Microglial Dysfunction Mediated by Pb and Amyloid Beta Peptides as a Possible Mechanism of Neurotoxicity Save
- Cowpea isoflavones enhance the osteoblast differentiation and antioxidant capacity in synergy with vitamin D and β-carotene: A mechanistic in vitro study Save
- M1 polarization induction by lead and amyloid peptides in microglial cells: Implications for neurodegeneration process Save
- Unveiling Potential Neurotoxic Mechansisms: Pb-Induced Activation of CDK5-p25 Signaling Axis in Alzheimer’s Disease Development, Emphasizing CDK5 Inhibition and Formation of Toxic p25 Species Save
- Heavy Metal Mediated Progressive Degeneration and Its Noxious Effects on Brain Microenvironment Save
- Exposure of combination of environmental pollutant, lead (Pb) and β-amyloid peptides causes mitochondrial dysfunction and oxidative stress in human neuronal cells Save
- Defective mitophagy and induction of apoptosis by the depleted levels of PINK1 and Parkin in Pb and β-amyloid peptide induced toxicity Save
- Mitochondria-Mediated Moderation of Apoptosis by EGCG in Cytotoxic Neuronal Cells Induced by Lead (Pb) and Amyloid Peptides Save