DS
Dr Moumita Sarkar
Imperial College London, MRC London Institute of Medical Sciences, Indian Institute of Chemical Biology CSIR, University of Calcutta
About
Studying mechanism of novel class of non-coding RNAs in cardiovascular disease and stem cell mediated cardiac regeneration.
Trained in molecular biology and cell signaling in stem cells and cancer. An independent thinker with a positive attitude towards scientific criticism, a productive contributor working in a team and well versed in scientific writing.
Passionate in cell signaling, in-vitro disease modeling and organoids.
Employment
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Imperial College London Research Associate2021 - Present
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MRC London Institute of Medical Sciences Postdoctoral Research Scientist2016 - 2022
Education
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Indian Institute of Chemical Biology CSIR PhD2010 - 2015
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University of Calcutta MSC2006 - 2008
Projects & Funding
Projects & funding information is unavailable.
Publications (12)
- Human Left Ventricle circRNA-miRNA-mRNA Network Analyses Reveal a Novel Proangiogenic Role for circNPHP1 Under Ischemic Conditions. Save
- Circular RNA regulatory role in pathological cardiac remodelling Save
- Human left ventricle circRNA-miRNA-mRNA network analyses reveals a novel proangiogenic role for circNPHP1 under ischemic conditions Save
- CNOT3 interacts with the Aurora B and MAPK/ERK kinases to promote survival of differentiating mesendodermal progenitor cells Save
- DEAD box RNA helicases crucial regulators of gene expression and oncogenesis Save
- The DEAD box protein p68: a novel coactivator of Stat3 in mediating oncogenesis Save
- The DEAD box protein p68: a crucial regulator of AKT/FOXO3a signaling axis in oncogenesis Save
- Corrigendum to “Exosome-mediated Delivery of the Intrinsic C-terminus Domain of PTEN Protects It From Proteasomal Degradation and Ablates Tumorigenesis” Save
- DEAD-box protein p68 is regulated by β-catenin/transcription factor 4 to maintain a positive feedback loop in control of breast cancer progression Save
- 2,2′-Diphenyl-3,3′-Diindolylmethane: A Potent Compound Induces Apoptosis in Breast Cancer Cells by Inhibiting EGFR Pathway Save