SN
Susanna S Ng
Also known as: Susanna S Ng
University Hospital Bonn, QIMR Berghofer Medical Research Institute, Griffith University School of Natural Sciences, Griffith University, Eskitis Institute for Drug Discovery
Research areas: Medicine · Oncology · T-cell and B-cell Immunology · Cancer Immunotherapy and Biomarkers · Malaria Research and Control
0000-0002-8494-7722
Employment
-
Universitätsklinikum Bonn Senior Postdoctoral Researcher2023 - Present
-
Universitätsklinikum Bonn Postdoctoral Researcher2020 - Present
-
QIMR Berghofer Medical Research Institute Research Officer2019 - 2020
-
QIMR Berghofer Medical Research Institute Research Assistant2018 - 2019
-
Griffith University Biomedical Sciences Laboratory Demonstrator2017 - 2017
-
Griffith University Immunology Tutor2016 - 2018
-
QIMR Berghofer Medical Research Institute Research Assistant2014 - 2015
-
QIMR Berghofer Medical Research Institute Research assistant2013 - 2014
Education
-
QIMR Berghofer Medical Research Institute PhD candidate2015 - 2019
-
Griffith University School of Natural Sciences PhD Candidate2015 - 2018
-
QIMR Berghofer Medical Research Institute Honours student2014 - 2014
-
Eskitis Institute for Drug Discovery Undergraduate student2013 - 2013
-
Lancaster University Exchange student2012 - 2012
-
Griffith University Bachelor of Biomedical Science with Honours2011 - 2014
-
University of Newcastle International Foundation Program2010 - 2011
Projects & Funding
-
Characterizing the role of non-canonical cytotoxic molecules in anti-tumor immunity Alexander von Humboldt Foundation2021 - 2023
Publications (39)
- Longitudinal spatial neutrophil profiling during ACT in murine melanoma reveals distinct lymph node infiltration patterns Save
- Longitudinal spatial profiling of neutrophils during adoptive T cell therapy in murine melanoma reveals distinct lymph node infiltration patterns across anatomical sites Save
- The Antibody–Drug Conjugate Sacituzumab Govitecan (IMMU-132) Represents a Potential Novel Therapeutic Strategy in Cholangiocarcinoma Save
- Flow cytometry protocol for cell death analysis in glioblastoma organoids: A technical note Save
- NKG7 is a Stable Marker of Cytotoxicity Across Immune Contexts and Within the Tumor Microenvironment Save
- Clonally expanded, targetable, natural killer-like NKG7 T cells seed the aged spinal cord to disrupt myeloid-dependent wound healing Save
- Type I Interferon Drives a Cellular State Inert to TCR‐Stimulation and Could Impede Effective T‐Cell Differentiation in Cancer Save
- Expression of the large amino acid transporter SLC7A5/LAT1 on immune cells is enhanced in primary sclerosing cholangitis-associated cholangiocarcinoma and correlates with poor prognosis in cholangiocarcinoma Save
- A molecular signature for IL-10–producing Th1 cells in protozoan parasitic diseases Save
- STING activation promotes autologous type I interferon–dependent development of type 1 regulatory T cells during malaria Save
- Human IL-10-producing Th1 cells exhibit a molecular signature distinct from Tr1 cells in malaria Save
- CD103+ Tissue Resident T-Lymphocytes Accumulate in Lung Metastases and Are Correlated with Poor Prognosis in ccRCC Save
- NKG7 Is Required for Optimal Antitumor T-cell Immunity Save
- Increased amphiregulin expression by CD4+ T cells from individuals with asymptomatic Leishmania donovani infection Save
- A pan-cancer fingerprint: common molecular denominators of the human tumor microenvironment Save
- Druggable epigenetic suppression of interferon-induced chemokine expression linked to MYCN amplification in neuroblastoma Save
- Systemic administration of IL‐33 induces a population of circulating KLRG1hi type 2 innate lymphoid cells and inhibits type 1 innate immunity against multiple myeloma Save
- IL-27 signalling regulates glycolysis in Th1 cells to limit immunopathology during infection Save
- The NK cell granule protein NKG7 regulates cytotoxic granule exocytosis and inflammation Save
- Type I Interferons Suppress Anti-parasitic Immunity and Can Be Targeted to Improve Treatment of Visceral Leishmaniasis. Save