Sukriti Baweja

Employment

• INSERM Postdoctoral Research Fellow
  2017 - 2018
• Institute of Liver and Biliary Sciences Senior Fellow
  2014 - Present
• University of Illinois at Chicago Postdoctoral Research Associate
  2011 - 2014

Education

• University of Delhi PhD
  2006 - 2010

Projects & Funding

Projects & funding information is unavailable.

Publications (27)

• Extracellular vesicles from hepatitis B patients serve as reservoir of hepatitis B virus DNA
  Journal of Viral Hepatitis 2019 DOI: 10.1111/jvh.12995
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• PAR2-Mediated cAMP Generation Suppresses TRPV4-Dependent Ca 2+ Signaling in Alveolar Macrophages to Resolve TLR4-Induced Inflammation
  Cell Reports 2019 DOI: 10.1016/j.celrep.2019.03.053
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• Coagulation failure is associated with bleeding events and clinical outcome during systemic inflammatory response and sepsis in acute-on-chronic liver failure: An observational cohort study
  Liver International 2019 DOI: 10.1111/liv.14034
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• Iron-Overload triggers ADAM-17 mediated inflammation in Severe Alcoholic Hepatitis
  Scientific Reports 2018 DOI: 10.1038/s41598-018-28483-x
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• Microvesicles in hepatic and peripheral vein can predict nonresponse to corticosteroid therapy in severe alcoholic hepatitis
  Alimentary Pharmacology and Therapeutics 2018 DOI: 10.1111/apt.14564
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• Molecular Ellipticity of Circulating Albumin-Bilirubin Complex Associates With Mortality in Patients With Severe Alcoholic Hepatitis
  Clinical Gastroenterology and Hepatology 2018 DOI: 10.1016/j.cgh.2017.11.022
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• Mucosal-associated invariant T cells are a profibrogenic immune cell population in the liver
  Nature Communications 2018 DOI: 10.1038/s41467-018-04450-y
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• Hyperoxidized albumin modulates neutrophils to induce oxidative stress and inflammation in severe alcoholic hepatitis.
  Hepatology (Baltimore, Md.) 2017 DOI: 10.1002/hep.28897
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• Functionally aberrant dendritic cell subsets and expression of DC-SIGN differentiate acute from chronic HBV infection.
  Hepatology international 2016 DOI: 10.1007/s12072-016-9763-0
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• Bone marrow stem cells and their niche components are adversely affected in advanced cirrhosis of the liver.
  Hepatology (Baltimore, Md.) 2016 DOI: 10.1002/hep.28754
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