0000-0002-9137-8150 Employment
Employment history is unavailable.
Education
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Bharathiar University Master of science2016 - 2018
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Assumption College Bachelor of science2013 - 2016
Projects & Funding
Projects & funding information is unavailable.
Publications (25)
- Repeated PM2.5 Inhalation Exposure Drives a Duration‐Dependent Transition From Mitochondrial Adaptation to Persistent Cardiac Toxicity in Rats Save
- Sodium thiosulfate mitigates PM2.5-induced cardiotoxicity by preservation of mitochondrial function. Save
- PM2.5 Induced Vascular and Myocardial Calcification Impairs Ischemia-reperfusion Tolerance via Mitochondrial Dysregulation. Save
- Investigating the temporal link between PM2.5 exposure and acceleration of myocardial ischemia reperfusion injury: Emphasizing the hazardous presence of metals in inhaled air Save
- PM2.5 toxicity in blood impairs cardiac redox balance and promotes mitochondrial dysfunction in rat heart that further aggravates ischemia reperfusion injury by modulating PI3K/AKT/mTOR/NF-kB signaling axis. Save
- PM2.5-Induced Declined Cardiac Tolerance to Ischemia-Reperfusion Injury Can Be Ameliorated by Hydrogen Sulfide Save
- Comparative evaluation of single and multiple exposure to PM2.5 in respirable air on cardiac physiology, structure and function in a Wistar rat model. Save
- PM2.5 Exposure-Linked Mitochondrial Dysfunction Negates SB216763-Mediated Cardio-Protection against Myocardial Ischemia-Reperfusion Injury. Save
- DNA hypomethylation by fisetin preserves mitochondria functional genes and contributes to the protection of I/R rat heart. Save
- Exposure to real ambient particulate matter inflicts cardiac electrophysiological disturbances, vascular calcification, and mitochondrial bioenergetics decline more than diesel particulate matter: consequential impact on myocardial ischemia-reperfusion i… Save