JK
Jonathan D Katz
University of California, Davis, Cincinnati Children's Hospital Medical Center, CNRS/INSERM - IGBMC, David Geffen School of Medicine at UCLA, School of Medicine, Washington University in St. Louis
0000-0002-9864-2268
Employment
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University of California, Davis Research Associate2025 - Present
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Cincinnati Children's Hospital Medical Center Professor2010 - 2024
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Cincinnati Children's Hospital Medical Center Associate Professor2000 - 2010
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School of Medicine, Washington University in St. Louis Assistant Professor1995 - 2000
Education
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CNRS/INSERM - IGBMC Post-Doctoral Fellow1990 - 1995
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David Geffen School of Medicine at UCLA PhD1986 - 1990
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UCLA BS1979 - 1984
Projects & Funding
Projects & funding information is unavailable.
Publications (49)
- Alloreactive memory CD4 T cells promote transplant rejection by engaging DCs to induce innate inflammation and CD8 T cell priming. Save
- Prior viral infection primes cross-reactive CD8+ T cells that respond to mouse heart allografts. Save
- Restoring tolerance to β-cells in Type 1 diabetes: Current and emerging strategies. Save
- T cells instruct myeloid cells to produce inflammasome-independent IL-1β and cause autoimmunity. Save
- Extending Remission and Reversing New-Onset Type 1 Diabetes by Targeted Ablation of Autoreactive T Cells. Save
- Dying to protect: cell death and the control of T-cell homeostasis. Save
- Manipulating DNA damage-response signaling for the treatment of immune-mediated diseases. Save
- Reversal of New-Onset Type 1 Diabetes With an Agonistic TLR4/MD-2 Monoclonal Antibody. Save
- Eliminating encephalitogenic T cells without undermining protective immunity. Save
- Etoposide selectively ablates activated T cells to control the immunoregulatory disorder hemophagocytic lymphohistiocytosis. Save