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Robin Andersson
University of Copenhagen, Uppsala University, Linköping University, Institute of Technology
0000-0003-1516-879X
About
Robin Andersson is Associate Professor of Bioinformatics and Deputy Head of Department for Research at the Department of Biology, University of Copenhagen. His research focuses on regulatory genomics, combining experimental genomics, computational biology, and machine learning to understand how noncoding DNA regulates gene expression and contributes to human disease. His laboratory develops methods to identify active regulatory elements, predict enhancer–gene interactions, uncover the sequence logic of gene regulation, and interpret the functional consequences of noncoding genetic variation.
Employment
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University of Copenhagen Associate professor2020 - Present
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University of Copenhagen Assistant Professor2015 - 2020
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University of Copenhagen Postdoctoral Research Fellow2011 - 2015
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Uppsala University PhD Student2005 - 2010
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Uppsala University Research Assistant2004 - 2004
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Linköping University, Institute of Technology TA in Imperative Programming in Ada 952003 - 2004
Education
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Uppsala University PhD Bioinformatics2005 - 2010
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Linköping University, Institute of Technology MSc Computer Science1999 - 2004
Projects & Funding
Projects & funding information is unavailable.
Publications (95)
- Metabolic disease-relevant stimuli unmask context-dependent genetic regulation of cardiometabolic loci in human adipocytes Save
- The human RNA-DNA interactome is cell type-specific and dynamic Save
- Time-resolved inference of gene regulatory networks underlying human cranial neural crest development suggests novel risk genes for orofacial clefting Save
- Mapping active cis-regulatory elements from transcription initiation events Save
- Mapping enhancer-gene regulatory interactions from single-cell data. Save
- Genome-wide rules of transcription factor cooperativity revealed through in silico binding site ablation Save
- Mapping enhancer-gene regulatory interactions from single-cell data Save
- Molecular convergence of risk variants for congenital heart defects leveraging a regulatory map of the human fetal heart Save
- Single cell variant to enhancer to gene map for coronary artery disease Save
- Publisher Correction: MYC activity at enhancers drives prognostic transcriptional programs through an epigenetic switch Save