SS
Satish Singh
CSIR-IHBT, The University of Arizona College of Medicine Phoenix, Institute of Microbial Technology CSIR, Guru Nanak Dev University, University of Arizona College of Medicine Phoenix
0000-0003-2963-8545
Employment
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CSIR-IHBT2021 - Present
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The University of Arizona College of Medicine Phoenix Research Scientist2020 - 2021
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University of Arizona College of Medicine Phoenix Postdoctoral Research Associate2017 - 2020
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University of Tennessee Health Science Center Postdoctoral fellow2012 - 2017
Education
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Institute of Microbial Technology CSIR Ph.D.2006 - 2012
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Guru Nanak Dev University MSc.2004 - 2006
Projects & Funding
Projects & funding information is unavailable.
Publications (15)
- Thromboembolic ischemic stroke induces neutrophil recruitment and matrix metalloproteinase-9 release to enhance ischemic brain injury Save
- Dispensability of a positively charged residue at Lys11 in staphylokinase for plasminogen activation Save
- Recent Pathophysiological Insights Are Advancing the Treatment of Venous Thromboembolism Save
- Targeting Fibrinolytic Inhibition for Venous Thromboembolism Treatment: Overview of an Emerging Therapeutic Approach Save
- Alpha2-Antiplasmin: The Devil You Don't Know in Cerebrovascular and Cardiovascular Disease. Save
- Integration of VEK-30 peptide enhances fibrinolytic properties of staphylokinase. Save
- Venous stasis-induced fibrinolysis prevents thrombosis in mice: role of α2-antiplasmin Save
- Matrix Metalloproteinase-9 Mediates the Deleterious Effects of α2-Antiplasmin on Blood-Brain Barrier Breakdown and Ischemic Brain Injury in Experimental Stroke. Save
- Releasing the Brakes on the Fibrinolytic System in Pulmonary Emboli: Unique Effects of Plasminogen Activation and α2-Antiplasmin Inactivation. Save
- Physiologic variations in blood plasminogen levels affect outcomes after acute cerebral thromboembolism in mice: a pathophysiologic role for microvascular thrombosis. Save