AO
Adam W. Olson
Van Andel Institute, Beckman Research Institute, City of Hope, University of Michigan, Miami University
Research areas: Medicine · Pulmonary and Respiratory Medicine · Prostate Cancer Treatment and Research · Sperm and Testicular Function · Wnt/β-catenin signaling in development and cancer
0000-0003-3118-0393
Employment
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Van Andel Institute Postdoctoral Fellow2026 - Present
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Beckman Research Institute, City of Hope2017 - 2020
Education
- 2020 - 2026
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Miami University B.S. in Engineering2013 - 2017
Projects & Funding
Projects & funding information is unavailable.
Publications (13)
- Amoeboid–mesenchymal transition and the proteolytic control of cancer invasion plasticity Save
- Amoeboid-Mesenchymal Transition and the Proteolytic Control of Cancer Invasion Plasticity Save
- Mmp14-dependent remodeling of the pericellular–dermal collagen interface governs fibroblast survival Save
- Androgen deprivation induces double-null prostate cancer via aberrant nuclear export and ribosomal biogenesis through HGF and Wnt activation Save
- Aberrant androgen action in prostatic progenitor cells induces oncogenesis and tumor development through IGF1 and Wnt axes Save
- Stromal androgen and hedgehog signaling regulates stem cell niches in pubertal prostate development Save
- Androgen action in cell fate and communication during prostate development at single-cell resolution. Save
- Androgen receptor with short polyglutamine tract preferably enhances Wnt/β-catenin-mediated prostatic tumorigenesis Save
- Loss of androgen signaling in mesenchymal sonic hedgehog responsive cells diminishes prostate development, growth, and regeneration Save
- Aberrant activation of hepatocyte growth factor/MET signaling promotes β-catenin–mediated prostatic tumorigenesis Save
- The comprehensive role of E-cadherin in maintaining prostatic epithelial integrity during oncogenic transformation and tumor progression Save
- A pivotal role of androgen signaling in Notch-responsive cells in prostate development, maturation, and regeneration Save
- Deletion of the p16INK4a tumor suppressor and expression of the androgen receptor induce sarcomatoid carcinomas with signet ring cells in the mouse prostate Save