Ravi Akundi

Employment

• South Asian University Assistant Professor
  2012 - Present
• University of Kentucky Post-doctoral Scholar
  2008 - 2012
• Yale University Post-doctoral Fellow
  2005 - 2008

Education

• Albert-Ludwigs-Universitat Freiburg Ph.D.
  2000 - 2005
• Indian Institute of Technology Bombay M.Sc.
  1998 - 2000

Projects & Funding

Projects & funding information is unavailable.

Publications (21)

• CD73-enriched extracellular vesicles reduce cyclooxygenase 2 (COX-2)-mediated inflammation in activated macrophages
  Biochemical and Biophysical Research Communications 2025 DOI: 10.1016/j.bbrc.2025.152845
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• P2Y12 receptor-mediated cyclooxygenase 2 (COX-2) expression enhances tumor cell progression in a mouse model of lymphoma
  Purinergic Signalling 2025 DOI: 10.1007/s11302-024-10057-4
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• P2Y6 receptor inhibition arrests tumor cell progression in a mouse lymphoma model
  Nucleosides, Nucleotides & Nucleic Acids 2025 DOI: 10.1080/15257770.2025.2584080
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• Imidazopyridazine Acetylcholinesterase Inhibitors Display Potent Anti-Proliferative Effects in the Human Neuroblastoma Cell-Line, IMR-32
  Molecules 2021 DOI: 10.3390/molecules26175319
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• Mutant Ataxin-3–Containing Aggregates (MATAGGs) in Spinocerebellar Ataxia Type 3: Dynamics of the Disorder
  Molecular Neurobiology 2021 DOI: 10.1007/s12035-021-02314-z
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• The anti-inflammatory effects of the 5-HT3 receptor antagonist tropisetron are mediated by the inhibition of p38 MAPK activation in primary human monocytes
  DOI: http://dx.doi.org/10.1016/j.bbi.2013.01.067
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• The anti-inflammatory effects of the 5-HT3 receptor antagonist tropisetron are mediated by the inhibition of p38 MAPK activation in primary human monocytes
  DOI: http://dx.doi.org/10.1016/j.intimp.2012.05.013
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• Substance P and valproic acid
  DOI: http://dx.doi.org/10.1046/j.1471-4159.2003.01802.x
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• Signal transduction pathways regulating cyclooxygenase-2 in lipopolysaccharide-activated primary rat microglia
  DOI: http://dx.doi.org/10.1002/glia.20198
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• Shared and Cell Type-Specific Mitochondrial Defects and Metabolic Adaptations in Primary Cells from PINK1-Deficient Mice
  DOI: http://dx.doi.org/10.1159/000345689
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