GK
Gregory Kelly
QIMR Berghofer Medical Research Institute
Research areas: Immunology and Microbiology · Immunology · Immune Cell Function and Interaction · Cancer Immunotherapy and Biomarkers · Epigenetics and DNA Methylation
0000-0003-4053-8342
Employment
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QIMR Berghofer Medical Research Institute Research Assistant2016 - Present
Education
Education history is unavailable.
Projects & Funding
Projects & funding information is unavailable.
Publications (39)
- Developing New Immunotherapy Approaches for Colorectal Cancer Save
- Data from FcRγ-Dependent NK Cell Licensing through CD244 Promotes Antitumor Immunity Save
- FcRγ-Dependent NK Cell Licensing through CD244 Promotes Antitumor Immunity Save
- Supplementary Figure S1 from FcRγ-Dependent NK Cell Licensing through CD244 Promotes Antitumor Immunity Save
- Supplementary Figure S10 from FcRγ-Dependent NK Cell Licensing through CD244 Promotes Antitumor Immunity Save
- Supplementary Figure S2 from FcRγ-Dependent NK Cell Licensing through CD244 Promotes Antitumor Immunity Save
- Supplementary Figure S3 from FcRγ-Dependent NK Cell Licensing through CD244 Promotes Antitumor Immunity Save
- Supplementary Figure S4 from FcRγ-Dependent NK Cell Licensing through CD244 Promotes Antitumor Immunity Save
- Supplementary Figure S5 from FcRγ-Dependent NK Cell Licensing through CD244 Promotes Antitumor Immunity Save
- Supplementary Figure S6 from FcRγ-Dependent NK Cell Licensing through CD244 Promotes Antitumor Immunity Save
- Supplementary Figure S7 from FcRγ-Dependent NK Cell Licensing through CD244 Promotes Antitumor Immunity Save
- Supplementary Figure S8 from FcRγ-Dependent NK Cell Licensing through CD244 Promotes Antitumor Immunity Save
- Supplementary Figure S9 from FcRγ-Dependent NK Cell Licensing through CD244 Promotes Antitumor Immunity Save
- Supplementary Table 1 from FcRγ-Dependent NK Cell Licensing through CD244 Promotes Antitumor Immunity Save
- Supplementary Table 2 from FcRγ-Dependent NK Cell Licensing through CD244 Promotes Antitumor Immunity Save
- Supplementary Table 3 from FcRγ-Dependent NK Cell Licensing through CD244 Promotes Antitumor Immunity Save
- Data from Combined Inhibition of G9a and EZH2 Suppresses Tumor Growth via Synergistic Induction of IL24-Mediated Apoptosis Save
- Data from Combined Inhibition of G9a and EZH2 Suppresses Tumor Growth via Synergistic Induction of IL24-Mediated Apoptosis Save
- Supplementary Data from Combined Inhibition of G9a and EZH2 Suppresses Tumor Growth via Synergistic Induction of IL24-Mediated Apoptosis Save
- Supplementary Data from Combined Inhibition of G9a and EZH2 Suppresses Tumor Growth via Synergistic Induction of IL24-Mediated Apoptosis Save