JG
José Afonso Guerra-Assunção
Erasmus MC, University of Cambridge, Instituto Gulbenkian de Ciência, Universidade de Lisboa Faculdade de Ciencias
Research areas: Medicine · Infectious Diseases · Genomics and Phylogenetic Studies · Acute Lymphoblastic Leukemia research · Tuberculosis Research and Epidemiology
0000-0001-6593-403X
Employment
- 2024 - Present
Education
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University of Cambridge Ph.D.2009 - 2012
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Instituto Gulbenkian de Ciencia PDBC 20072007 - 2012
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Universidade de Lisboa Faculdade de Ciencias Microbiology and Genetics2003 - 2007
Projects & Funding
Projects & funding information is unavailable.
Publications (82)
- Disease-Attenuated Pneumococcal Biosynthesis Gene Mutants Invade the Mucosal Epithelium and Induce Innate Immunity Save
- The CSF transcriptome in adults with pneumococcal meningitis reveals compartmentalised host inflammatory responses associated with mortality Save
- The Streptococcus pneumoniae transcriptome in patient cerebrospinal fluid identifies novel virulence factors required for meningitis Save
- Mitochondrial dsRNA from B-ALL cells stimulates mesenchymal stromal cells to become cancer associated fibroblasts Save
- Essential role of proline synthesis and the one-carbon metabolism pathways for systemic virulence ofStreptococcus pneumoniae Save
- SARS-CoV-2 viral dynamics and population pharmacokinetics of favipiravir in combination with lopinavir-ritonavir in patients with COVID-19 Save
- Disease-attenuated pneumococcal biosynthesis gene mutants invade the mucosal epithelium and induce innate immunity Save
- Genomic investigations of unexplained acute hepatitis in children Save
- Persistent SARS-CoV-2 PCR Positivity Despite Anti-viral Treatment in Immunodeficient Patients Save
- Data from EZH2-Deficient T-cell Acute Lymphoblastic Leukemia Is Sensitized to CHK1 Inhibition through Enhanced Replication Stress Save
- Data from EZH2-Deficient T-cell Acute Lymphoblastic Leukemia Is Sensitized to CHK1 Inhibition through Enhanced Replication Stress Save
- Supplementary Figures and Legends from EZH2-Deficient T-cell Acute Lymphoblastic Leukemia Is Sensitized to CHK1 Inhibition through Enhanced Replication Stress Save
- Supplementary Figures and Legends from EZH2-Deficient T-cell Acute Lymphoblastic Leukemia Is Sensitized to CHK1 Inhibition through Enhanced Replication Stress Save
- Supplementary Table 1 from EZH2-Deficient T-cell Acute Lymphoblastic Leukemia Is Sensitized to CHK1 Inhibition through Enhanced Replication Stress Save
- Supplementary Table 1 from EZH2-Deficient T-cell Acute Lymphoblastic Leukemia Is Sensitized to CHK1 Inhibition through Enhanced Replication Stress Save
- Supplementary Table 2 from EZH2-Deficient T-cell Acute Lymphoblastic Leukemia Is Sensitized to CHK1 Inhibition through Enhanced Replication Stress Save
- Supplementary Table 2 from EZH2-Deficient T-cell Acute Lymphoblastic Leukemia Is Sensitized to CHK1 Inhibition through Enhanced Replication Stress Save
- Supplementary Table 3 from EZH2-Deficient T-cell Acute Lymphoblastic Leukemia Is Sensitized to CHK1 Inhibition through Enhanced Replication Stress Save
- Supplementary Table 3 from EZH2-Deficient T-cell Acute Lymphoblastic Leukemia Is Sensitized to CHK1 Inhibition through Enhanced Replication Stress Save
- Supplementary information from EZH2-Deficient T-cell Acute Lymphoblastic Leukemia Is Sensitized to CHK1 Inhibition through Enhanced Replication Stress Save