CM
COSTANZA MONTAGNA
University of Rome Tor Vergata
Research areas: Biochemistry, Genetics and Molecular Biology · Molecular Biology · Redox biology and oxidative stress · Cancer, Hypoxia, and Metabolism · Nitric Oxide and Endothelin Effects
Employment
Employment history is unavailable.
Education
- 2011 - 2015
Projects & Funding
Projects & funding information is unavailable.
Publications (20)
- The Bright and Dark Sides of Nitric Oxide in Neurodegenerative Diseases Save
- “Feeding the Rhythm”—Effects of Food and Nutrients on Daily Cortisol Secretion: From Molecular Mechanisms to Clinical Impact Save
- Duchenne Muscular Dystrophy: Integrating Current Clinical Practice with Future Therapeutic and Diagnostic Horizons Save
- Duchenne Muscular Dystrophy: Integrating Current Clinical Practice with Future Therapeutic and Diagnostic Horizons Save
- Looking at denitrosylation to understand the myogenesis gone awry theory of rhabdomyosarcoma Save
- Autophagy guards tendon homeostasis Save
- AMBRA1 regulates cyclin D to guard S-phase entry and genomic integrity. Save
- Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1. Save
- Comparison of Tenocyte Populations from the Core and Periphery of Equine Tendons. Save
- When S-nitrosylation gets to mitochondria: from signaling to age-related diseases. Save
- nNOS/GSNOR interaction contributes to skeletal muscle differentiation and homeostasis. Save
- S-nitrosylation drives cell senescence and aging in mammals by controlling mitochondrial dynamics and mitophagy. Save
- S-nitrosylation of the Mitochondrial Chaperone TRAP1 Sensitizes Hepatocellular Carcinoma Cells to Inhibitors of Succinate Dehydrogenase. Save
- To eat, or NOt to eat: S-nitrosylation signaling in autophagy. Save
- S-Nitrosoglutathione Reductase Plays Opposite Roles in SH-SY5Y Models of Parkinson's Disease and Amyotrophic Lateral Sclerosis. Save
- Extremely Low Frequency Magnetic Field (ELF-MF) Exposure Sensitizes SH-SY5Y Cells to the Pro-Parkinson's Disease Toxin MPP(.). Save
- S-nitrosoglutathione reductase deficiency-induced S-nitrosylation results in neuromuscular dysfunction. Save
- S-nitrosation and ubiquitin-proteasome system interplay in neuromuscular disorders. Save
- Reticulon1-C modulates protein disulphide isomerase function. Save
- Established Principles and Emerging Concepts on the Interplay between Mitochondrial Physiology and S-(De)nitrosylation: Implications in Cancer and Neurodegeneration. Save