TS
Teresa Sadras
Olivia Newton-John Cancer Wellness & Research Centre
Research areas: Medicine · Hematology · Acute Lymphoblastic Leukemia research · Lymphoma Diagnosis and Treatment · Acute Myeloid Leukemia Research
0000-0001-9048-9580
Employment
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Olivia Newton-John Cancer Wellness & Research Centre Laboratory Head
Education
Education history is unavailable.
Projects & Funding
Projects & funding information is unavailable.
Publications (24)
- Programmable and selective silencing of oncogenic fusion transcripts by CRISPR-Cas13b via breakpoint sequence recognition Save
- IRF2BP2::JAK2 Defines a Novel Kinase-Activating Fusion in Pediatric T-Cell Acute Lymphoblastic Leukemia. Save
- CEACAM1 as a mediator of B-cell receptor signaling in mantle cell lymphoma. Save
- Generative AI and digital twins: shaping a paradigm shift from precision to truly personalized medicine. Save
- A novel TRKB-activating internal tandem duplication characterizes a new mechanism of receptor tyrosine kinase activation. Save
- Principles of CRISPR-Cas13 mismatch intolerance enable selective silencing of point-mutated oncogenic RNA with single-base precision. Save
- A novel MYB::PAIP1 oncogenic fusion in pediatric blastic plasmacytoid dendritic cell neoplasm (BPDCN) is dependent on BCL2 expression and is sensitive to venetoclax. Save
- TALLSorts: a T-cell acute lymphoblastic leukemia subtype classifier using RNA-seq expression data. Save
- Unusual PDGFRB fusion reveals novel mechanism of kinase activation in Ph-like B-ALL. Save
- SFPQ-ABL1 and BCR-ABL1 use different signaling networks to drive B-cell acute lymphoblastic leukemia. Save
- JAFFAL: detecting fusion genes with long-read transcriptome sequencing. Save
- PON2 subverts metabolic gatekeeper functions in B cells to promote leukemogenesis. Save
- Metabolic Gatekeepers of Pathological B Cell Activation. Save
- IFITM3 functions as a PIP3 scaffold to amplify PI3K signalling in B cells. Save
- Signalling input from divergent pathways subverts B cell transformation. Save
- MEF2D Fusions Drive Oncogenic Pre-BCR Signaling in B-ALL. Save
- B-Cell-Specific Diversion of Glucose Carbon Utilization Reveals a Unique Vulnerability in B Cell Malignancies. Save
- Pre-B acute lymphoblastic leukaemia recurrent fusion, EP300-ZNF384, is associated with a distinct gene expression. Save
- High prevalence of relapse in children with Philadelphia-like acute lymphoblastic leukemia despite risk-adapted treatment. Save
- Differential expression of MUC4, GPR110 and IL2RA defines two groups of CRLF2-rearranged acute lymphoblastic leukemia patients with distinct secondary lesions. Save